3.6C Neuropsychology

Anomic fluent aphasia

Broca's aphasia

Transcortical non-fluent aphasia

Global non-fluent aphasia

Alexia

Agraphia

Word deafness

Behandeling aphasia

Motorfunctie: posterior cortex

Motorfunctie: prefrontal cortex

Motorfunctie: premotorcortex (in FL tussen PFC en motorcortex)

Motorfunctie: primaire motor cortex (M1)

Brainstem

Basal ganglia

Cerebellum

Hypothalamus

Hippocampus

Langetermijngeheugen declaratief (expliciet)

Langetermijngeheugen niet-declaratief (impliciet)

Kortetermijngeheugen

Functie: primary motor cortex, premotor cortex, prefrontal cortex

How can we characterize prosopagnosia, and which brain area is damaged when people are affected by this disease?

How can we characterize ideational apraxia and which area of the brain is damaged when people are affected by this disease?

According to the paper of Heutik et al., visual agnosia can be treated with restorative and compensatory methods. What is the goal of these treatments?

According to the paper of Park et al., non-invasive brain stimulation can improve rehabilitative treatment for apraxia. How does that work?

According to the paper of Lunven et al., which are the best predictors for the recovery from contralateral neglect?

Gerstmann syndrome is a neurological disorder characterized by a cluster of symptoms related to difficulties in various aspects of cognition and motor function. This syndrome is typically associated with damage to the left parietal lobe. What is a typical characteristic?

lint's syndrome typically results from damage to the posterior parietal cortex. What is a typical characteristic of this syndrome?

The primary motor cortex (M1) plays an important role in motor control? What is the specific role and where is this area located in the brain?

Volgorde: Arcuate Fasciculus, Broca, Wernicke en functie

In nonfluent aphasia which brain area is typical dysfunctional and what is a typical symptom?

What brain area is typically dysfunctional in anomic (amnesic) aphasia and what is a typical symptom?

In the study of emotion we use electrodermal (EDA) and cardiac (heart rate) responses. What does EDA reflect?

Both lesions of the amygdala and of the ventromedial prefrontal cortex (vmPFC) lead to reduced capacity to recognize emotional face expressions (Vaidya). Despite the similar deficits on the behavioral level, the underlying mechanisms are quite distinct. What is the difference in mechanism?

According to the somatic marker hypothesis, in what way are subcortical regions such as amygdala and hippocampus involved in decision-making?

According to the theory of LeDoux two separate neural pathways can be used to activate the Amygdala? How can these two be distinguished?

Brain areas belonging to the limbic system are associated with processing of emotion. Which brain areas belong to the limbic system?

How can Huntington's disease and Parkinson's disease be distinguished from each other?

Parkinson's disease can be characterized by negative and positive symptoms. Two of these negative symptoms are:

Which of the following is not a characteristic of Alzheimer's disease?

Which statement about fetal alcohol spectrum disorder (FASD) is not correct? Children with FASD may present with:

Which of the following characteristics is commonly observed in Parkinson's patients?

What can be done to prevent Alzheimer's disease?

What is seen as the underlying cause of Multiple Sclerosis?

Which factors have been linked to the etiology of multiple sclerosis?

In Tourette's syndrome we can distinguish three phases. The first phase is characterized by:

How can we distinguish between fetal alcohol syndrome (FAS) and ADHD in terms of executive functions?

Functions: 1) OFC, 2) thalamus, 3) hippocampus, 4) cingulate cortex

Which is NOT a characteristic of attention?

Which brain area is involved in conflict monitoring and emotional responses?

Which executive function is affected by damage to the dorsolateral prefrontal cortex (dlPFC)?

What component of the attentional control network proposed by Posner is responsible for maintaining attention over time?

What is the correct order of the time-organized stages through which memories are stored?

How does the involvement of the hippocampus change when a memory is being consolidated?

Which statement about the Modal Model by Atkinson & Shiffrin (1968) is correct?

Alzheimer’s disease is characterized by widespread atrophy of the cortex. In which lobe is the atrophy most severe?